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Increased expression of interleukin-22 in patients with giant cell arteritis

Rheumatology Sep 12, 2017

Zerbini A, et al. – In this study, the authors presented the hypothesis that interleukin-22 (IL-22) might be induced in giant cell arteritis (GCA) and might be involved in disease pathogenesis. They explained that in patients with GCA, IL-22 was increased and affected the viability and gene expression of arterial cells. These findings indicated its potential role in the disease pathogenesis.
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