Scheibe F, Neumann WJ, Lange C, et al. - In this study, the clinical spectrum, neuroimaging results, therapy, and prognosis of these debilitating post‐hypoxic sequelae were explored. Between January 2007 and September 2018, researchers conducted a retrospective study including a total of 72 patients from the neurological intensive care unit at a university hospital, who were diagnosed with hypoxic‐ischaemic encephalopathy after cardiac arrest. They screened clinical records for the occurrence of post‐hypoxic movement disorders and chronic post‐hypoxic myoclonus. They further examined affected patients for used neuroprognostic tests, administered therapy and treatment response, and the outcome of these movement disorders and neurological function. The data exhibited that post‐hypoxic movement disorders correlated with pronounced basal ganglia dysfunction might be efficiently controlled by levomepromazine or intrathecal baclofen. It was indicated that their occurrence might be an indicator for a more unfavourable, but often not devastating, neurological outcome.